Thank you. I am so swamped with material that I probably can’t read it. It does sound like a plausible explanation for acute respiratory illnesses and the apparent contagion that resists experimental replication.
Presumably people vary in their abiiity to regulate airway surface liquid under stressful atmospheric conditions.
I have presented an alternate lung physiology in my Substack article titled: we breathe air not oxygen
I say winter presents lungs with dry air and dehydration is the major factor that induces cold and flu symptoms
Dryness being our weak point
I can discard the whole oxygen/carbon dioxide gaseous exchange and show a logical physiological process of hydration at the alveoli sacs.
I hope you can take the time to read it
Thank you. I am so swamped with material that I probably can’t read it. It does sound like a plausible explanation for acute respiratory illnesses and the apparent contagion that resists experimental replication.
Presumably people vary in their abiiity to regulate airway surface liquid under stressful atmospheric conditions.
The airway mucosa must condition the incoming air with salt and moisture
100% humidity at the alveoli
The surfactant produced at the alveoli makes bubbles
The bubbles burst upon the alveoli sac capillaries - spraying saline water upon the passing RBCs - the blood cells hydrate
The state changes of the RBCs is from dehydration to hydration- contracted to expanded - dense/dark to bright
The red light monitoring is recording hydration by measuring the light from the RBCs nothing to do with oxygen
There’s more, my article takes you through step by step
It’s a paradigm shift in understanding lung physiology