"Furthermore, regulatory T-cell activation can non-specifically dampen immune response to other pathogens, leading to increased incidence of secondary infections."
It bears remembering that the virtue signalers at WHO, in making the case to rename monkeypox, made two key assertions: there are a number of genomes that don't fit cleanly in …
"Furthermore, regulatory T-cell activation can non-specifically dampen immune response to other pathogens, leading to increased incidence of secondary infections."
It bears remembering that the virtue signalers at WHO, in making the case to rename monkeypox, made two key assertions: there are a number of genomes that don't fit cleanly in the West African clade, and monkeypox is spreading human-to-human in Western Europe, a region with high inoculation uptake.
"Furthermore, regulatory T-cell activation can non-specifically dampen immune response to other pathogens, leading to increased incidence of secondary infections."
It bears remembering that the virtue signalers at WHO, in making the case to rename monkeypox, made two key assertions: there are a number of genomes that don't fit cleanly in the West African clade, and monkeypox is spreading human-to-human in Western Europe, a region with high inoculation uptake.
https://allfactsmatter.substack.com/p/the-faucists-dilemma-politically
If the inoculations dampen broad immune system response, that goes a long way in explaining the increased pathogenicity.
That's still a speculation, but if the immune system damage is comprehensive in the human host, the monkeypox outbreak gets much easier to explain.